Fig. 7. (  A ) Effects of 15 μm amyloid β  42(Aβ  42) peptide with and without halothane and isoflurane at 1 and 2 minimum alveolar concentration on lactate dehydrogenase release over 72 h in pheochromocytoma cells. Bars represent the mean ± sem (>3 wells, three experiments) magnitude of change from untreated control wells. *  P < 0.05 as compared with control; †  P < 0.05 as compared with control and the equivalent anesthetic concentration alone; ‡  P < 0.05 as compared with control, the equivalent anesthetic concentration, and the Aβ  42peptide alone (analysis of variance and  post hoc Tukey-Kramer testing). (  B ) The analogous results for propofol and ethanol. 

Fig. 7. (  A ) Effects of 15 μm amyloid β  42(Aβ  42) peptide with and without halothane and isoflurane at 1 and 2 minimum alveolar concentration on lactate dehydrogenase release over 72 h in pheochromocytoma cells. Bars represent the mean ± sem (>3 wells, three experiments) magnitude of change from untreated control wells. *  P < 0.05 as compared with control; †  P < 0.05 as compared with control and the equivalent anesthetic concentration alone; ‡  P < 0.05 as compared with control, the equivalent anesthetic concentration, and the Aβ  42peptide alone (analysis of variance and  post hoc Tukey-Kramer testing). (  B ) The analogous results for propofol and ethanol. 

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