Fig. 6. Summarized data for effects of Gö 6976 (10 μm), rottlerin (10 μm), protein kinase C (PKC) ϵ V1-2 (10 μm), and myristoylated (Myrst) PKCζ inhibitor peptide (10 μm) on the propofol-induced (30 μm) increase in cell shortening during α1a-adrenoreceptor activation with phenylephrine (10 μm). Results are expressed as percent of control (Ctrl), which represents the twitch amplitude achieved with phenylephrine in the absence of the inhibitors, set to 100%. *  P < 0.05 compared with control value. n = at least 3 cells with each inhibitor from 7 hearts. 

Fig. 6. Summarized data for effects of Gö 6976 (10 μm), rottlerin (10 μm), protein kinase C (PKC) ϵ V1-2 (10 μm), and myristoylated (Myrst) PKCζ inhibitor peptide (10 μm) on the propofol-induced (30 μm) increase in cell shortening during α1a-adrenoreceptor activation with phenylephrine (10 μm). Results are expressed as percent of control (Ctrl), which represents the twitch amplitude achieved with phenylephrine in the absence of the inhibitors, set to 100%. *  P < 0.05 compared with control value. n = at least 3 cells with each inhibitor from 7 hearts. 

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