Figure 2. (A) Left ventricular end-systolic pressure (LVESP), (B) left ventricular end-diastolic pressure (LVEDP), (C) peak positive rate of change in left ventricular pressure (LV +dp/dt), and (D) ratio of LV +dp/dt to peak negative rate of change in left ventricular pressure (LV -dp/dt) in malignant hyperthermia-susceptible (MHS) swine (filled circles; n = 12) and non-MHS (nMHS) swine (open circles; n = 8) during baseline recordings (t = 0) and after 90 min of halothane exposure (1% inspired) in addition to succinylcholine (3 mg *symbol* kg sup -1 intravenously 15 min after the start of halothane exposure). *P < 0.05 versus baseline; triangles = P < 0.05 MHS versus nMHS.

Figure 2. (A) Left ventricular end-systolic pressure (LVESP), (B) left ventricular end-diastolic pressure (LVEDP), (C) peak positive rate of change in left ventricular pressure (LV +dp/dt), and (D) ratio of LV +dp/dt to peak negative rate of change in left ventricular pressure (LV -dp/dt) in malignant hyperthermia-susceptible (MHS) swine (filled circles; n = 12) and non-MHS (nMHS) swine (open circles; n = 8) during baseline recordings (t = 0) and after 90 min of halothane exposure (1% inspired) in addition to succinylcholine (3 mg *symbol* kg sup -1 intravenously 15 min after the start of halothane exposure). *P < 0.05 versus baseline; triangles = P < 0.05 MHS versus nMHS.

Close Modal
This Feature Is Available To Subscribers Only

or Create an Account

Close Modal
Close Modal