Fig. 1. Photomicrographs of dopamine-β-hydroxylase (DβH)– and phosphorylated cyclic adenosine monophosphate response element binding protein (pCREB)–immunoreactive neurons in brainstem slices from normal rats. Brainstem slices treated with vehicle (  A–C ), 100 μm gabapentin (GBP;  D–F ), and 50 μm kainate (  G–I ) for 30 min were stained with antibodies for DβH (  green ) and pCREB (  red ). Note that GBP (  G ) and kainate (  I ) increased pCREB immunoreactivity in DβH-immunoreactive neurons.  Scale bar = 100 μm. 

Fig. 1. Photomicrographs of dopamine-β-hydroxylase (DβH)– and phosphorylated cyclic adenosine monophosphate response element binding protein (pCREB)–immunoreactive neurons in brainstem slices from normal rats. Brainstem slices treated with vehicle (  A–C ), 100 μm gabapentin (GBP;  D–F ), and 50 μm kainate (  G–I ) for 30 min were stained with antibodies for DβH (  green ) and pCREB (  red ). Note that GBP (  G ) and kainate (  I ) increased pCREB immunoreactivity in DβH-immunoreactive neurons.  Scale bar = 100 μm. 

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